By Icon Health Publications

ISBN-10: 0497000423

ISBN-13: 9780497000424

ISBN-10: 1417541334

ISBN-13: 9781417541331

It is a 3-in-1 reference publication. It provides a whole clinical dictionary overlaying thousands of phrases and expressions in terms of aldosterone. It additionally provides huge lists of bibliographic citations. eventually, it offers details to clients on the right way to replace their wisdom utilizing numerous net assets. The booklet is designed for physicians, clinical scholars getting ready for Board examinations, scientific researchers, and sufferers who are looking to familiarize yourself with study devoted to aldosterone. in case your time is effective, this e-book is for you. First, you won't waste time looking the net whereas lacking loads of appropriate info. moment, the e-book additionally saves you time indexing and defining entries. eventually, you won't waste money and time printing enormous quantities of web content.

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Additional info for Aldosterone - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References

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Intrarenal AnglI excess and/or activation of AnglI-generating elements; and 2C. increased renal vascular reactivity to AnglI. AIM 3 will test the hypothesis that effects of MPR and/or Body-Size Excess are mediated by the AT1 receptor. Subaims will test effects of in vivo AT1R blockade or placebo 3A. on hemodynamic and 3B. on AnglI contractile signaling via classic and EGFR-transactivation routes (using renal vascular reactivity and activated vs total signaling protein abundance in fresh vascular/cortical tissues).

Genomic DNA will be extracted. We propose to evaluate the association and linkage between BP, HTN and genes of the RAAS (ACE, AGT and angiotensin II receptor type 1), epithelial sodium channel beta subunit, endothelial nitric oxide synthase, CRP, endothelin 1, and beta2adrenergic receptor. We will evaluate single nucleotide polymorphisms (SNP) variations in candidate genes and determine if variation in each SNP and in constructed haplotype, explain variations in BP and HTN risk. In addition, DNA will be stored for future application to conduct genome-wide scan with the goal of identifying novel genomic regions in linkage and association with BP and HTN in AA.

Upregulation of other transport pathways will also be examined using thiazides to block NaCI cotransport and angiotensin receptor antagonists to prevent upregulation of Na/H exchange. We will also assess the roles of two putative signaling pathways for aldosterone secretion under these conditions, increased renin activity and elevated plasma K. We will explore the mechanisms of pathological upregulation seen in the Liddle's Syndrome form of human hypertension. This will be done using mice in which the channel subunit B-ENaC has been truncated to mimic the human mutation.

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Aldosterone - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by Icon Health Publications


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